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Volume 27, Issue 1, pp. 15-23
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History
- Received April 14, 2020
- Revision received June 17, 2020
- Accepted July 31, 2020
- Published first August 5, 2020.
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- Previous version (August 5, 2020 - 06:02).
- Previous version (December 2, 2020 - 05:56).
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©2020 American Association for Cancer Research.
Author Information
- 1Myeloma Service, Department of Medicine, Memorial Sloan Kettering Cancer Center, New York, New York.
- 2Perlmutter Cancer Center, New York University Langone Health, New York, New York.
- ↵*Corresponding Authors:
Francesco Maura, Memorial Sloan Kettering Cancer Center, 1275 York Ave, New York, NY 10044. Phone: 212-639-5126; Fax: 646-277-7116; E-mail: mauraf{at}mskcc.org; Gareth J. Morgan, Myeloma Program, Perlmutter Cancer Center, New York University Langone Health, 522, First Avenue, New York, NY 10016. Phone: 212-263-0649; E-mail: Gareth.Morgan{at}nyulangone.org; and Ola Landgren, Myeloma Service, Department of Medicine, Memorial Sloan Kettering Cancer Center, 1275 York Ave, New York, NY 10065. Phone: 212-639-5126; Fax: 646-277-7116; E-mail: Landgrec{at}mskcc.org
January 2021
Volume 27, Issue 1
Volume 27, Issue 1
Designing Evolutionary-based Interception Strategies to Block the Transition from Precursor Phases to Multiple Myeloma
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- Article
- Abstract
- Introduction
- Multiple Myeloma as a Model to Understand Cancer Initiation and Progression
- Mutational Signatures Driving Progression of Early-Premalignant Phases to Multiple Myeloma
- The Temporal Relationship of Mutational Signatures and Driver Events
- Insights from Functional Genomics
- Building Models to Understand the Bionetworks Active in Precursor Disease Phases
- Manipulating Multiple Myeloma Ecology to Intercept Disease Transformation to Multiple Myeloma
- Authors' Disclosures
- Acknowledgments
- Footnotes
- References
- Figures & Data
- Info & Metrics
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